ysplasia of the hip in babies is relatively common, especially in girls. It is more likely in babies who are breech births, in twins, or ones who have a reduced volume of amniotic fluid in the womb. It also runs in families. Medical staff dealing with babies are always fanatically careful about testing babies to make sure their hips are stable after they are born. Most of the babies with dysplasia are picked up in the first few weeks using a routine clinical test where, with the baby on her back and legs flexed at the hip, the examiner attempts to push the head of the femur out of the hip socket.
It used to be called ‘Congenital Dislocation of the Hip’ (CDH). Nowadays it is now called ‘Developmental Dysplasia of the Hip’ (DDH), because we realise there are some babies who examine normally at birth, but over the early months of life develop an abnormal hip joint, whose socket becomes flattened and unstable. This latter group is caused by an inborn genetic factor or inappropriate posturing of the legs over the first months (See last paragraph).
For the first year a baby’s hip joint is mostly made of cartilage, which is mouldable. Beyond one year of age it turns to bone, which is not. We therefore wish to diagnose dysplasia while the joint is still cartilage so treatment can fix it completely. If treatment is delayed until after the joint turns to bone such treatment is never completely satisfactory. The joint usually remains abnormal and the patient may end up with a limp or, at the least, develop early osteoarthritis requiring a hip replacement in early adult life.